Biblio
Found 1700 results
Author [ Keyword] Title Type Year Filters: First Letter Of Last Name is V [Clear All Filters]
“Developmentally stable whole-brain volume reductions and developmentally sensitive caudate and putamen volume alterations in those with attention-deficit/hyperactivity disorder and their unaffected siblings.”, JAMA Psychiatry, vol. 72, no. 5, pp. 490-9, 2015.
, “Heritability of the shape of subcortical brain structures in the general population.”, Nat Commun, vol. 7, p. 13738, 2016.
, “Heritability of the shape of subcortical brain structures in the general population.”, Nat Commun, vol. 7, p. 13738, 2016.
, “Heritability of the shape of subcortical brain structures in the general population.”, Nat Commun, vol. 7, p. 13738, 2016.
, “Alpha-T-catenin is expressed in human brain and interacts with the Wnt signaling pathway but is not responsible for linkage to chromosome 10 in Alzheimer's disease.”, Neuromolecular Med, vol. 5, no. 2, pp. 133-46, 2004.
, “Alpha-T-catenin is expressed in human brain and interacts with the Wnt signaling pathway but is not responsible for linkage to chromosome 10 in Alzheimer's disease.”, Neuromolecular Med, vol. 5, no. 2, pp. 133-46, 2004.
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“Rarity of the Alzheimer disease-protective APP A673T variant in the United States.”, JAMA Neurol, vol. 72, no. 2, pp. 209-16, 2015.
, “Rarity of the Alzheimer disease-protective APP A673T variant in the United States.”, JAMA Neurol, vol. 72, no. 2, pp. 209-16, 2015.
, “Rarity of the Alzheimer disease-protective APP A673T variant in the United States.”, JAMA Neurol, vol. 72, no. 2, pp. 209-16, 2015.
, “Rarity of the Alzheimer disease-protective APP A673T variant in the United States.”, JAMA Neurol, vol. 72, no. 2, pp. 209-16, 2015.
, “Rarity of the Alzheimer disease-protective APP A673T variant in the United States.”, JAMA Neurol, vol. 72, no. 2, pp. 209-16, 2015.
, “Revealing the role of glutathione S-transferase omega in age-at-onset of Alzheimer and Parkinson diseases.”, Neurobiol Aging, vol. 27, no. 8, pp. 1087-93, 2006.
, “Rarity of the Alzheimer disease-protective APP A673T variant in the United States.”, JAMA Neurol, vol. 72, no. 2, pp. 209-16, 2015.
, “Heritability of the shape of subcortical brain structures in the general population.”, Nat Commun, vol. 7, p. 13738, 2016.
, “Heritability of the shape of subcortical brain structures in the general population.”, Nat Commun, vol. 7, p. 13738, 2016.
, “Heritability of the shape of subcortical brain structures in the general population.”, Nat Commun, vol. 7, p. 13738, 2016.
, “Heritability of the shape of subcortical brain structures in the general population.”, Nat Commun, vol. 7, p. 13738, 2016.
, “Heritability of the shape of subcortical brain structures in the general population.”, Nat Commun, vol. 7, p. 13738, 2016.
, “Heritability of the shape of subcortical brain structures in the general population.”, Nat Commun, vol. 7, p. 13738, 2016.
, “Transcriptomic Changes Due to Cytoplasmic TDP-43 Expression Reveal Dysregulation of Histone Transcripts and Nuclear Chromatin.”, PLoS One, vol. 10, no. 10, p. e0141836, 2015.
, “GWAS of cerebrospinal fluid tau levels identifies risk variants for Alzheimer's disease.”, Neuron, vol. 78, no. 2, pp. 256-68, 2013.
, “A common haplotype lowers PU.1 expression in myeloid cells and delays onset of Alzheimer's disease.”, Nat Neurosci, vol. 20, no. 8, pp. 1052-1061, 2017.
, “A common haplotype lowers PU.1 expression in myeloid cells and delays onset of Alzheimer's disease.”, Nat Neurosci, vol. 20, no. 8, pp. 1052-1061, 2017.
, “A common haplotype lowers PU.1 expression in myeloid cells and delays onset of Alzheimer's disease.”, Nat Neurosci, vol. 20, no. 8, pp. 1052-1061, 2017.
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